
“Just eat when you’re hungry and stop when you’re full.”
Sounds simple, right?
That’s the promise behind intuitive eating—the new wellness buzzword that encourages us to listen to our bodies, honor our hunger cues, and ditch diet culture for good. And in theory, it’s a beautiful idea. I love the idea of trusting your body again. But here’s the part no one is saying out loud:
If your hormones are out of whack—especially your insulin and appetite hormones—your body is lying to you.
Let’s break this down.
Insulin Resistance Hijacks Appetite Hormones
Your hunger and fullness aren’t just emotional. They’re biological. You have actual hormones whose job it is to regulate how much you eat:
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Ghrelin tells you you’re hungry.
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Leptin tells you you’re full.
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GLP-1 helps you feel satisfied and slows digestion.
But when you have insulin resistance, this whole system gets flipped upside down. Chronically high insulin levels make your brain resistant to leptin, so you don’t hear the “I’m full” signal. They ramp up ghrelin, so you feel hungry even when you shouldn’t. And they suppress GLP-1, making it harder to feel satisfied after eating.
Your body doesn’t feel “off”—it feels like it’s in survival mode. It tells you to eat again… and again… and again.
And it’s not your fault. These hormones are supposed to work in harmony. But insulin resistance scrambles the signal. That’s why telling someone with insulin resistance to “eat intuitively” is a joke.
Here’s how serious this issue has become: We now have an entire class of medications—GLP-1 receptor agonists like Ozempic and Wegovy—designed to override the body’s broken appetite system. They flood your system with GLP-1 because your body struggles to make enough on its own to feel full.
But why are your appetite signals messed up in the first place? Because insulin resistance is disrupting your appetite hormones. Instead of looking at why those signals are off, we’re trying to medicate around them. And while those meds have their place, the root cause is still being ignored.
Why You Feel So Different From “Naturally Thin” People
If you’ve ever looked at someone who seems effortlessly thin and thought:
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How do they eat so little?
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Why are they not hungry all the time?
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Do they know some secret I don’t?
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Are they just stronger than me? More disciplined?
Here’s the truth: they’re probably just less hungry. Not because they’re better than you—because their appetite hormones are working like they’re supposed to.
Their body sends clear signals. They eat when they’re hungry and stop when they’re full—and they’re not battling cravings or a growling stomach two hours later.
Meanwhile, if you’re insulin resistant, your hunger cues are completely out of sync. Your brain is getting told to eat even when your body has plenty of fuel stored. So yeah, intuitive eating is hard when your own hormones are sabotaging you.
Here’s the Real Path to Intuitive Eating
You want to be able to trust your body again? To eat when you’re hungry, stop when you’re full, and not feel like food is running your life?
You have to fix insulin first.
The only way to truly eat intuitively is to first repair the hormonal signals that drive hunger and fullness. And that means lowering insulin.
That’s where a Low Insulin Lifestyle comes in. It’s not about restriction or punishment. It’s about eating in a way that brings your hormones back into balance—so your body can finally send the right signals at the right time.
Only then does intuitive eating actually become intuitive.
Bottom Line
If your hormones are screaming “Eat! Eat! Eat!” all day long, intuitive eating won’t feel empowering—it’ll feel like a battle you keep losing.
Before you can trust your hunger cues, you have to teach your hormones how to whisper again.
And the only way to do that is by bringing insulin down.
So no, you’re not broken.
You’re just trying to “eat intuitively” with a hormone system that’s been hijacked.
Balance insulin first—and your body will start talking sense again.
References
Verdich C, et al. The role of postprandial releases of insulin and incretin hormones in meal-induced satiety–effect of obesity and weight reduction. Int J Obes Relat Metab Disord. 2001. Read more
Abdul-Ghani M, et al. Managing insulin resistance: the forgotten pathophysiological component of type 2 diabetes. Lancet Diabetes Endocrinol. 2024. Read more